Parainfluenza virus type 3

Common Name(s)

Parainfluenza virus type 3

Description for this condition is not yet available.
 

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Condition Specific Organizations

Following organizations serve the condition "Parainfluenza virus type 3" for support, advocacy or research.

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Advocacy and Support Organizations

 

Condition Specific Organizations

Following organizations serve the condition "Parainfluenza virus type 3" for support, advocacy or research.

There are currently no organizations listed in Disease InfoSearch that support this condition. Create a listing.

 

 

General Support Organizations

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Scientific Literature

Articles from the PubMed Database

Research articles describe the outcome of a single study. They are the published results of original research.
The terms "Parainfluenza virus type 3" returned 103 free, full-text research articles on human participants. First 3 results:

A leucine residue in the C terminus of human parainfluenza virus type 3 matrix protein is essential for efficient virus-like particle and virion release.
 

Author(s): Guangyuan Zhang, Shengwei Zhang, Binbin Ding, Xiaodan Yang, Longyun Chen, Qin Yan, Yanliang Jiang, Yi Zhong, Mingzhou Chen

Journal: J. Virol.. 2014 Nov;88(22):13173-88.

 

Paramyxovirus particles, like other enveloped virus particles, are formed by budding from membranes of infected cells, and matrix (M) proteins are critical for this process. To identify the M protein important for this process, we have characterized the budding of the human parainfluenza ...

Last Updated: 21 Oct 2014

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Terminal sialic acid linkages determine different cell infectivities of human parainfluenza virus type 1 and type 3.
 

Author(s): Keijo Fukushima, Tadanobu Takahashi, Seigo Ito, Masahiro Takaguchi, Maiko Takano, Yuuki Kurebayashi, Kenta Oishi, Akira Minami, Tatsuya Kato, Enoch Y Park, Hidekazu Nishimura, Toru Takimoto, Takashi Suzuki

Journal: Virology. 2014 Sep;464-465():424-31.

 

Human parainfluenza virus type 1 (hPIV1) and type 3 (hPIV3) initiate infection by sialic acid binding. Here, we investigated sialic acid linkage specificities for binding and infection of hPIV1 and hPIV3 by using sialic acid linkage-modified cells treated with sialidases or sialyltransferases. ...

Last Updated: 6 Sep 2014

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Phosphoprotein of human parainfluenza virus type 3 blocks autophagosome-lysosome fusion to increase virus production.
 

Author(s): Binbin Ding, Guangyuan Zhang, Xiaodan Yang, Shengwei Zhang, Longyun Chen, Qin Yan, Mengyao Xu, Amiya K Banerjee, Mingzhou Chen

Journal: Cell Host Microbe. 2014 May;15(5):564-77.

 

Autophagy is a multistep process in which cytoplasmic components, including invading pathogens, are captured by autophagosomes that subsequently fuse with degradative lysosomes. Negative-strand RNA viruses, including paramyxoviruses, have been shown to alter autophagy, but the molecular ...

Last Updated: 16 May 2014

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Reviews from the PubMed Database

Review articles summarize what is currently known about a disease. They discuss research previously published by others.
The terms "Parainfluenza virus type 3" returned 1 free, full-text review articles on human participants. First 3 results:

Medical burden of respiratory syncytial virus and parainfluenza virus type 3 infection among US children. Implications for design of vaccine trials.
 

Author(s): Min-Shi Lee, Robert E Walker, Paul M Mendelman

Journal: Hum Vaccin. ;1(1):6-11.

 

Respiratory syncytial virus (RSV) and parainfluenza virus type 3 (PIV3) are two leading causes of lower respiratory illness (LRI) in infants. Many efforts have been directed to develop vaccines against these two viruses. Licensure of new vaccines includes three phases of clinical ...

Last Updated: 13 Oct 2006

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Symptoms, Diagnosis, and Treatment

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Clinical Trial Information This information is provided by ClinicalTrials.gov

There are currently no open clinical trials for this condition.