Glomerulonephritis

Common Name(s)

Glomerulonephritis

Glomerulonephritis is a type of kidney disease in which there is inflammation of the glomeruli, the tiny filters that remove excess waste and fluids from the blood.  It may be acute or chronic (coming on gradually), and may occur on its own (primary) or be caused by another condition (secondary). Signs and symptoms may depend on the type and cause of the condition and may include blood in the urine; foamy urine; swelling of the face, eyes, or other body parts; abdominal pain; high blood pressure; fatigue; and/or other symptoms. If the condition is severe or prolonged, the kidneys can become damaged.  It may be caused by a variety of infections and diseases, and sometimes the cause is unknown. Treatment depends on the cause, type and severity of the condition.
 

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Advocacy and Support Organizations

 

Condition Specific Organizations

Following organizations serve the condition "Glomerulonephritis" for support, advocacy or research.

There are currently no organizations listed in Disease InfoSearch that support this condition. Create a listing.

 

 

General Support Organizations

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Scientific Literature

Articles from the PubMed Database

Research articles describe the outcome of a single study. They are the published results of original research.
The terms "Glomerulonephritis" returned 1208 free, full-text research articles on human participants. First 3 results:

Glomerulonephritis and granulomatous vasculitis in kidney as a complication of the use of BRAF and MEK inhibitors in the treatment of metastatic melanoma: A case report.
 

Author(s): Mehdi Maanaoui, Camille Saint-Jacques, Viviane Gnemmi, Marie Frimat, Arnaud Lionet, Marc Hazzan, Christian Noël, François Provot

Journal: Medicine (Baltimore). 2017 Jun;96(25):e7196.

 

BRAF and MEK inhibitors have significantly improved the prognosis of metastatic melanoma, by inhibiting both the mitogen-activated protein kinase (MAP-kinase) pathway. They are associated with infrequent adverse kidney events. Most of these are related to the use of BRAF inhibitors ...

Last Updated: 22 Jun 2017

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Different types of glomerulonephritis associated with the dysregulation of the complement alternative pathway in 2 brothers: A case report.
 

Author(s): Pei Chen, Li Zhu, Feng Yu, Sha-Sha Han, Si-Jun Meng, Wei-Yi Guo, Hong Zhang, Yan Song

Journal: Medicine (Baltimore). 2017 Jun;96(24):e7144.

 

C3 glomerulonephritis (C3GN) and complement-mediated hemolytic uremic syndrome (HUS) both result from the abnormal regulation of the complement system. A significant number of patients with C3GN or complement-mediated HUS have mutations of more than 1 complement protein. This discovery ...

Last Updated: 14 Jun 2017

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Concurrent IgG4-related tubulointerstitial nephritis and IgG4 myeloperoxidase-anti-neutrophil cytoplasmic antibody positive crescentic glomerulonephritis: A case report.
 

Author(s): Tao Su, Li Yang, Zhao Cui, Su-Xia Wang, Ming-Hui Zhao

Journal: Medicine (Baltimore). 2017 May;96(20):e6707.

 

IgG4-related disease (IgG4-RD) is a newly recognized systemic disease. The typical pathological finding in the kidney is abundant IgG4-positive plasma cell infiltration with characteristic storiform fibrosis in the interstitium. Antibodies of the IgG4 subclass have been linked to ...

Last Updated: 17 May 2017

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Reviews from the PubMed Database

Review articles summarize what is currently known about a disease. They discuss research previously published by others.
The terms "Glomerulonephritis" returned 133 free, full-text review articles on human participants. First 3 results:

CD4(+) T Cell Fate in Glomerulonephritis: A Tale of Th1, Th17, and Novel Treg Subtypes.
 

Author(s): Christan F Krebs, Oliver M Steinmetz

Journal: Mediators Inflamm.. 2016 ;2016():5393894.

 

Multiple studies have identified CD4(+) T cells as central players of glomerulonephritis (GN). Cells of the Th1 and Th17 responses cause renal tissue damage, while Tregs mediate protection. Recently, a high degree of plasticity among these T cell lineages was proposed. During inflammation, ...

Last Updated: 15 Dec 2016

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C3 glomerulonephritis in multiple myeloma: A case report and literature review.
 

Author(s): Guang Yin, Zhen Cheng, Cai-Hong Zeng, Zhi-Hong Liu

Journal: Medicine (Baltimore). 2016 Sep;95(37):e4843.

 

C3 glomerulonephritis (C3 GN) is a recently defined entity characterized by predominant C3 deposition in glomeruli due to abnormal activation of the alternative pathway of complement system. C3 GN has been reported to be associated with several systemic diseases. However, the association ...

Last Updated: 16 Sep 2016

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Spontaneous remission of membranous glomerulonephritis with successful fetal outcome: A case report and literature review.
 

Author(s): Yan-Mei Huang, Hui-Rong Zhou, Ling Zhang, Ke-Ke Yang, Jiang-Xi Luo, Hai-Lu Zhao

Journal: Medicine (Baltimore). 2016 Jun;95(26):e4022.

 

Membranous glomerulonephritis (MGN) represents an immunologically mediated disease characterized by deposition of immune complexes in the glomerular subepithelial space. Persistent proteinuria at diagnosis predicts poor prognosis. Pregnancy with MGN is a risk of fetal loss and may ...

Last Updated: 2 Jul 2016

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Clinical Trial Information This information is provided by ClinicalTrials.gov

ACE-inhibitors in Extracapillary Glomerulonephritis
 

Status: Recruiting

Condition Summary: Extracapillary Glomerulonephritis

 

Last Updated: 19 Jul 2017

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Effect of Rituximab in Treatment of Membranoproliferative Glomerulonephritis
 

Status: Not yet recruiting

Condition Summary: Membranoproliferative Glomerulonephritis

 

Last Updated: 15 Jun 2017

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Acute Post Streptococcal Glomerulonephritis
 

Status: Not yet recruiting

Condition Summary: PSGN - Post-Streptococcal Glomerulonephritis

 

Last Updated: 13 Jun 2017

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